By integrating these strategies, we can move toward a new generation of interventions that not only slow disease progression but may also mitigate the impact of Alzheimers disease across diverse genetic backgrounds, heralding a future with more effective, targeted treatments
The role of autophagy in resistance to targeted therapies
Elevated glutathione renders cells more resistant against, e.g., mitochondrial inhibition, oxidative, or proteasomal stress
It should be noted that the fold changes of these redox defects are much more pronounced than that of glutamate level decrease in SLC7A11 high cancer cells under glucose starvation, and thereby correlate better with the rapid and dramatic cell death observed in these cells upon glucose starvation (Liu et al., 2020)
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