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Microvascular dysfunction induced by sepsis is closely related to vascular endothelial cell apoptosis, which is mediated by caspases and iNOS/NAPDH oxidase pathways

(9) also noted an altered Tfh cell subset composition associated with increased levels of Tfh2 and Tfh17 cells but decreased levels of Tfh1 and Tfh17.1 cells

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The cytokine addition prolonged the CLL cells survival and reduced the absolute ROS production