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As such, the results suggest that the use of Ferr-1 partially reverses the apoptotic and necroptotic effects of anisomycin on HuOCSCs

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Specific endonucleases XPG and ERCC1/XPF cleave the lesion, and after removal of a 2432 nucleotide fragment, the remaining single-strand gap is filled by the replication machinery and the resulting nick is sealed by ligase I or ligase III (de Boer and Hoeijmakers, 2011)

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