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Their prevalence spans from 10% in GSD 0a [16] and GSD III [17] to 17% in GSD IXb [18], 27% in GSD Ia [19], 30% in GSD IXa [18], 38% in GSD Ib [19], 52% in GSD VI [20], 57% in GSD IV patients [21] and up to 90% of GSD XI individuals [22,23,24,25]

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In the first step of this cycle, the SAM-dependent methylases (EC 2.1.1) that use SAM as a substrate produce S -adenosyl homocysteine as a product

As a result, this approach allows examination of the biological roles of a stabilized form of galectin-1 without introducing the confounding variables that can occur when typical soluble reducing agents are employed

doi: 10.1016/j.prp.2023.154372

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