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Prevention therapies and treatments The development of CIPN is likely multifactorial and involves several mechanisms as discussed above, although it is currently unclear which effect(s) initiate the pathological cascade leading to neuronal dysfunction

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At the molecular levels, our data showed that the protective effect of DHEA is mediated by stimulating Bcl-2 levels and upregulation/activation of SIRT1 signaling, including deacetylation of Nrf2, NF-B p65, p53, and FOXO3a