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It supports detox pathways, maintains redox balance, and promotes healthy immune and cellular function.*

For example, mTOR promotes the induction of c-MYC and HIF-1, which supports aerobic glycolysis and glutamine catabolism

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Next, we determined the protective effect of four catechol estrogens (2-OH-E 1 , 2-OH-E 2 , 4-OH-E 1 and 4-OH-E 2

Additionally, glutamine metabolism supports the mitochondrial functions and cellular redox metabolic capacity of AML cells, resulting in the adverse prognosis of AML patients carrying the FLT3-ITD mutation