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Superoxide dismutase (SOD), an antioxidant enzyme, converts superoxide anion (O2) to hydrogen peroxide (H 2 O 2 ) which in turn is reduced to water by catalase [28]

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This was primarily due to GA pretreatment enhancing antioxidant defenses, inhibiting the functions of neurotoxicity-related proteins (Bax, TNF- and caspase-3), and improving cerebral I/R injury in rats, thereby exhibiting neuroprotective properties (Farbood et al., 2013

Divalent metal transporter 1 (DMT1) regulates the release of Fe 2+ into the labile iron pool (LIP) for further utilization, and extra Fe 2+ is retained in ferritin