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Specifically, the PTEN-induced kinase 1 (PINK1)/Parkin pathway is a classic mechanism that activates mitophagy by mediating the recognition of damaged mitochondria

Because of the lethal potential of cyanide toxicity, most organisms have developed effective detoxification mechanisms that classically require sulfur donors, primarily sulfur-containing amino acids, to convert CN into thiocyanate (SCN), a putatively less toxic molecule 9

Previous studies suggested that ferroptosis is executed by peroxidizing PUFA-chains-containing phospholipids (PL), especially phosphatidylethanolamines (PE) and phosphatidylcholines (PC) [44], while the lysophospholipids (LP) increase upon ferroptosis inducer treatments as the product of enzymatic cleavage of the oxidized PUFA tail [45, 46]

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Comprehensive analysis supports the fundamental difference between senescence and eryptosis, evidenced primarily by much faster clearance of eryptotic cells compared to senescent erythrocytes [78]

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