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R.de BarrosAODSHelal-NetoE.Dos SantosS

The primary aim of this study is to elucidate the molecular mechanisms underlying CP-induced ovarian injury, with a focus on oxidative stress, inflammatory signaling, and apoptotic pathways, focusing on nuclear factor-kappa B (NF-B), nuclear factor erythroid-2-related factor 2/heme oxygenase-1 (Nrf2/HO-1), Toll-like receptor 4 (TLR4), nucleotide-binding oligomerization domain-like receptor family pyrin domain-containing 3 (NLRP3) inflammasome, silent information regulator-1 (SIRT1), and other signaling pathways involved in the pathogenesis of ovarian injury caused by CP

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aeruginosa 89 from a TSA overnight culture was grown in 40 ml of Nutrient Broth II (Sifin Diagnostics GmbH, Berlin, DE) for 4 h at 37C at 140 r/min

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