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Figure 8 summarizes the main points of this paper, where we claim that excessive levels of deuterium in the mitochondria due to gut dysbiosis may be the driving force that ultimately results in cardiolipin oxidation and migration to the outer membrane, following its accumulation of deuterium bound to the bis-allylic carbon atoms

While there are plenty of discussions on the Internet, there is very little human clinical evidence

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and they were listed in the Merck Medical Manual until the mid-1970s

To further confirm that DON can induce ferroptosis in granulosa cells, we co-cultured granulosa cells with DON and different concentrations of ferroptosis inhibitor Fer-1 (0.110 M)