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Augmenting brain B 12 with fingolimod or potentially related molecules could enhance both current and future MS therapies. In their paper, the team at Sanford Burnham Prebys, with collaborators at University of Southern California, Juntendo University in Japan, Tokyo University of Pharmacy and Life Sciences and State University of New York, focused on the molecular functioning of FTY720 or fingolimod (Gilenya), a sphingosine 1-phosphate (S1P) receptor modulator that suppresses distribution of T and B immune cells errantly attacking the brains of MS patients

When a compound has such a narrow target, the potential for off-target side effects is inherently limited

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J Pain Palliat Care Pharmacother

However, there is no evidence for this hypothesis

The main ingredients help to breakdown and metabolism of fat in the body