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Juxtaposed with the Lurcher mice, where degradation mainly starts due to the mutated GluD2 receptor, CCAS patients have a variety of cerebellar disorder etiologies

In addition, OMP-54F28, an FZD8 decoy receptor, can competitively bind to Wnts to block this pathway, and its efficacy in combination with sorafenib was tested in patients with advanced HCC in a phase I clinical trial (NCT02069145)