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doi: 10.1007/s10096-018-3237-3

[DOI] [PubMed] [Google Scholar] 48.Ricci E., Al Beitawi S., Cipriani S., Candiani M., Chiaffarino F., Vigan P., Noli S., Parazzini F

A similar effect was found in AK BSO where tGSH in the blood, kidney, and liver was 61% (AK Wtr 0.26 0.02 mg/mL

Of relevance here is that hypoxia-activated IGF-1R signaling not only induces SDF1/CXCR4 expression and activation but also transactivates CXCR4 signaling to collectively enhance hypoxia-mediated GSC migration (Kuo et al., 2018)

10.1007/s11356-019-06224-5 28 ChenX.WangZ.ZhuG.NordbergG

57 Conversely, excessive ROS overwhelm the cells antioxidant defenses, triggering oxidative damage and, ultimately, programmed cell death