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Receptors quickly deactivate upon unbinding and washing away of the agonist ligand

The persistence of this state ultimately leads to neuronal damage and synaptic function loss through multiple mechanisms

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For instance, insulin is a peptide

SLC7A11 is a critical regulatory target involved in VRK2 regulating the GSH metabolic synthesis pathway Aforementioned data indicated that VRK2-deficient PC cells are vulnerable to GSH inhibition, this prompts us to explore the link between VRK2 and GSH metabolic synthesis pathway

Regulation of G protein-coupled receptor kinases and arrestins during receptor desensitization