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Hormonal alterations Since stress has been described as a potential trigger for ME/CFS (422) and cause of symptom flare-ups (423, 424), it is likely that the HPA axis- the neurobiological stress system- may be implicated in ME/CFS (425, 426), as well as abnormalities in growth hormone (GH) secretion and dysfunctional adrenergic metabolism (255)

The binding of H1 peptides to the tetrameric c-Myc-92 is the mechanism of inhibition [43]

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Activated B cells differentiate into plasma cells, producing pathogenic autoantibodies (anti-endothelial cell antibodies, anti-AT1R/ETAR antibodies, anti-BMPR2 antibodies)