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reduction, alkylation, and digestion)

In addition, nutrient deprivation in the PDAC tumor microenvironment (TME) [31] may drive KRAS -mutated cancer cells to be more dependent on lysosomal nutrient scavenging pathways, such as macropinocytosis [27] and autophagy [32], to recycle energy and biosynthetic building blocks from the microenvironment [16]

Furthermore, N2L protects HT22 cells from RSL3-induced ferroptosis by inhibiting activation of the JNK/ERK signaling pathway, diminishing lipid peroxidation through the restoration of GPX4 expression, and the reduction of ACSL4 and COX-2 protein levels [79]

erinaceus defensive function, showing the inhibition of inducible NO synthase (iNOS), phosphorylation of p38 MAPK and CHOP, and the reduction of acute inflammatory cytokine levels [46]

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