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Fibroblast activation protein CAR-Ms have been demonstrated to alleviate myocardial fibrosis and maintain cardiac function, presenting a novel therapeutic strategy for myocardial ischemia-reperfusion injury and other CVDs characterized by a fibrotic phenotype (112)

FIGURE 8 Additional stability and degradation escape of CD44v9-xCT complex is carried out through interaction with ubiquitin thioesterase/otubain-1 (OTUB1), a deubiquitinating enzyme (DUB) that plays a vital role in regulating protein stability by removing ubiquitin molecules from target proteins, thereby preventing their degradation by the proteasome and as a result mediating ferroptosis (Liu et al., 2019

Curr Neurol Neurosci Rep 11:274282

Heterologous pyc gene expression under various natural and engineered promoters in Escherichia coli for improved succinate production

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