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55 stated that doxorubicin-induced cardiotoxicity caused a remarkable decline of GSH levels, whereas LPO levels significantly increased

Considering that NF-B activity is directly linked to the LPS-induced expression of iNOS and secretion of NO, it can be concluded that AM404 most likely modulates the secretion of NO from LPS-stimulated microglia through its inhibitory action on the NF-B pathway [70,71]

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These microorganisms of the intestinal tract also reinforce the barrier function of the intestinal lining, decreasing the passage of pathogenic bacteria or antigens from the gut into the bloodstream [3]