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Previous studies have demonstrated that SalB effectively inhibits the binding of CD36 to its ligand, thereby mitigating its detrimental effects in atherosclerosis and inflammation (88, 89)

In a subsequent, 16-week study in biopsy-confirmed F1F3 NASH patients, approximately half of whom were type 2 diabetes patients, efruxifermin improved glycemic control with significant reductions in HbA1c at doses up to 70 mg weekly