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GPX4 and alzheimers disease GPX4, an antioxidant enzyme, is highly expressed in NCOA4 deficient mice (Yoo et al., 2012)

The above experiment was repeated in presence of 10 l of liver supernatant (360 g protein ml -1 ) that contained glutathione transferase and small amount of GSH

Pretreatment with the RIPK1 inhibitors necrostatin-1 or necrostatin-1s or the MLKL inhibitor necrosulfonamide prevented phosphorylation of RIPK1 after DC661 (1 M) treatment in melanoma cells (Figure 2G) but failed to rescue DC661-induced cytotoxicity in melanoma cells (Figure 2H) or colon cancer or pancreatic cancer cells (Supplemental Figure 2G)

Known activators and blockers of SmTRPM PZQ were shown to increase or decrease PZQ sensitivity respectively