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Key cytoprotective enzymes and molecules involved in ROS scavenging, including superoxide dismutase (SOD), glutathione peroxidase (GPx), catalase (CAT), and glutathione (GSH), play central roles in maintaining redox homeostasis

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They regulate blood-brain exchanges [1] at a time when the cerebral vascular network is not fully developed and parenchymal blood flow is low [2, 3]

Lyons KE

The UPR-PERK pathway is not a promising therapeutic target for mutant SOD1-induced ALS

Levels of glutathione fluctuate due to nutrient concentrations, exercise, oxidative stress and ageing