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The first description of the disease was carried out in 1906 by a German psychiatrist and neurologist, Alois Alzheimer, when exploring the brain tissue of a patient, Auguste Deter, who had died from a mental illness unknown at that time [2, 3]

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Dihydropteridine reductase deficiency associated with severe neurologic disease and mild hyperphenylalaninemia

Three derivatives of the D-Nle-X-Ile-(6)amino-hexanoic amide, where X is: Cys, Trp, or Met were examined to determine whether the percent of inhibition of dimerization and the binding affinity for each compound for HGF could be correlated to in vitro cellular activity, namely inhibition of MDCK cell scattering

Key Takeaway: The proposed mechanism involves activation of the hepatocyte growth factor (HGF) / c-Met receptor pathway, supporting synaptic plasticity How Does Dihexa Compare to Other Cognitive Interventions