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Inhalation of PM exacerbates respiratory symptoms in patients with chronic airway diseases, but the mechanisms underlying this response remain unclear

Antihypertensive drugs such as ACE inhibitors act to interfere with conversion of angiotensin I to the biologically active form angiotensin II, and angiotensin receptor blockers (ARBs) act as antagonists at the AT 1 receptor thus preventing angiotensin II binding

The release of bromelain in tumour-bearing mice by pH-sensitive NPs increased the hydrolysis of the ECM favoring the penetration of the anticancer drug (164)

Studies have shown that SOD1 dysfunction is closely related to a variety of diseases, such as hypertension, liver disease and tumor 56

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In line with reduced weight loss and adipocyte size, plasma non-esterified fatty acid (NEFA) levels were significantly higher than in HFD-IF mice (Fig