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However, the 0.8 and 1 mM GDC-treated monolayers showed a significantly increased extracellular activity ( P 0.05)

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it was shown that miR-31 is upregulated in AF and depletes neuronal NOS and dystrophin, leading to shortened APD [162]

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Additionally, the isotopologue profiles in the lung and serum were the most closely matched (Supplementary Fig

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