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Figure 2 Table 1 Multitarget mechanisms by which capsaicin modulates cardiometabolic syndrome related pathophysiological processes

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In ECs treated with TGF-, excessive glycolysis diminishes cytoplasmic NADPH production, and triggers a compensatory efflux of mitochondrial NADPH through isocitrate/-ketoglutarate shuttle [49]

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Prevalence and prognosis significance of cardiovascular disease in cancer patients: a population-based study

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