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Liu CG, Cui XL, Wei ZG, Guo JS

The loss of liver mass in recovering DTA-CCl 4 -injured Lrat YFP-Col1a1-DTR mice was attributed to the marked shrinkage of hepatocyte cytoplasm, decrease of hepatocyte proliferation and abnormal hepatocyte differentiation in the absence of HSCs

To further confirm iron-catalyzed H 2 S production in the blood and plasma as non-enzymatic, we utilized several pharmacologic, biochemical, and genetic tools to rule out CBS, CGL, and/or 3-MST activity

& Gewecke, M

Learn more: #PIPsignaling #Phosphoinositides #LipidResearch #DrugDiscovery To view or add a comment, sign in PUBLICATION ALERT Recent shifts of checkpoint inhibitors to the neoadjuvant setting allows to deeply investigate the mechanism-of-action of immune therapy with molecular imaging AND tissue analysis

You can also compare the broader approach in our Liposomal Delivery guide