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18 CaoC.YudinY.BikardY.ChenW.LiuT.LiH.et al

Given the omnipresence of oxidative stress in biological systems, bacteria have evolved sophisticated mechanisms to mitigate its harmful effects, particularly in metal-rich environments

and finally, overexpression of a phospho-dead mutant of TBC1D4 (mutation of the Akt phosphosites S318, S588, T642, and S751 to Ala) impeded insulin-stimulated GLUT4 traffic in a dominant-negative manner [96]