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Specifically, L2-Cmu was engineered as a human/murine chimera (i.e., reverse chimeric antibody), such that the complementary-determining regions (CDRs) were engineered into a new framework in which the entirety of the variable regions are human and the constant regions are mouse

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This follow the observations that vascular co-option is a mechanism of resistance [32] but the angiogenic status of a tumour can change during progression in both ways [3,29]

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