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Following either the process of aging in older individuals, uremic milieu, or prolonged in vitro expansion, MSCs encounter cellular senescence [199]

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Collectively, maternal obesity appears to overstimulate placental nutrient-sensing networks (mTOR, insulin/IGF-1, PPAR) while inhibiting energy homeostasis pathways (AMPK, SIRT1), leading to excessive nutrient transfer and long-term metabolic consequences for the offspring [49,53,55,56,57,58]