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The relative mRNA levels of these genes were measured using the gr knockout strain with the wild type as the control or using the gr overexpression strain with the wild type carrying vacant pJRD215 plasmid as the control

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These findings suggest that impaired methylation reactions and/or protein synthesis may not be the main mechanisms underlying moderate hyperhomocysteinemia-induced vascular disease

Abnormal intracellular lipid accumulation induces mitochondrial dysfunction, reduces mitochondrial quantity, and further promotes lipid accumulation [81]