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Genetic predisposition, co-existing mental health disorders, prior substance abuse, and environmental influences such as peer pressure or high academic and professional demands are all potential contributors

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Previous studies suggest that the downstream products of NLRP3 inflammasomes such as IL-1 or IL-18 are associated with the pathophysiology of smoke-driven chronic obstructive pulmonary disease (COPD) although direct evidence to link NLRP3 protein with the disease is sparse

I am hoping it is a blown fuse