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These molecules act as signals that recruit inflammatory cells, including neutrophils and macrophages, to the site of cell death, exacerbating local inflammation and potentially contributing to miscarriage ( 5.3 Evidence of ferroptosis in RM pathophysiology Emerging studies support the involvement of ferroptosis in RM, highlighting its potential role in pathophysiology through various lines of evidence

This local iron overload in DSCs triggers ferroptosis through reduced GSH and glutathione peroxidase four levels with the concomitant increase in lipid and cellular ROS

Additionally, it plays a role in protecting spermatozoa from DNA damage and preserving acrosome integrity, which is essential for successful fertilization [35]

doi: 10.1016/S0009-8981(03)00200-6