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in the early and middle stages of heat stress, D-dimer and soluble thrombomodulin levels increase in patients, while they decrease during the most severe stage of organ injury, suggesting possible anti-fibrinolytic thrombosis [32]

Instead, degeneration and/or scarring of the tendon due to long term wear and tear develop and are the cause of the problem

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He also described the small particulate component of the cytoplasm (which became known as 'Palade's granules' or ribosomes)