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CHAC1-mediated GSH degradation reduces S-glutathionylation of ARF6 at cysteine 90, thereby inactivating ARF6 and enhancing ferroptotic cell death ( Similarly, CHAC1 is upregulated in various kidney disease models ( CHAC1 upregulation was induced by cysteine and methionine deprivation, treatment with erastin and exposure to tunicamycin

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44 However, due to the heterogeneity of clinical studies, drug dosing and formulation, as well as difficulty in identifying the target population among those with COPD, the implementation of carbocisteine or NAC in the treatment of COPD has been limited