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The term also refers to the mechanism behind these changes: functionally relevant alterations to the genome that do not involve mutations in the nucleotide sequence

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More fat burned = glycogen spared = longer time to fatigue Especially beneficial for CrossFit, endurance athletes, and metabolic conditioning MOTS-c improves physical performance by regulating mitochondrial respiration and metabolic substrate use. Reynolds et al., Nature Communications Supports Anti-Aging and Stress Resistance Emerging research suggests that MOTSc acts like a mitochondrial stress hormone , improving longevity pathways similar to caloric restriction

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Now, [s]omehave looked on the exercise-induced increase in free radical production as a paradox. Why would an apparently healthy act (exercise) [lead] to detrimental effects through damage to various molecules and tissues