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Intracellular trafficking of FcRn and its cargo is controlled by Rab GTPases, specifically Rab4 and Rab11, which modulate the recycling pathway by separating sorting endosomes from transport end exocytotic vesicles [47]

In a patient with normal B12 status, injecting more of it does not create surplus energy or burn fat

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Early intervention might focus on modulating the initial TGF- response to prevent excessive fibroblast activation and ECM production

For exercise performance and VO2 max improvement, the evidence from human exercise studies suggests that MOTS-c's acute rise following high-intensity training is part of the adaptive signal driving mitochondrial biogenesis and cardiovascular remodeling

However, studies in aging models are rare, and existing studies mainly focus on ER neuroprotective effects [73]