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Fever plus mitochondrial disease could be risk factors for autistic regression

Taken together, these data demonstrate that PYL and GID1 act as antagonistic switches on DWA1, and DWA1 functions as a receptor-gated degradation channel that integrates the two hormone signals into a single outputRGL2 stability

doi: 10.1126/science.1224159

In conclusion, our data suggest that the treatment with the antioxidant peptide Epitalon enhanced impaired wound healing processes in HG-injured ARPE-19 cells by inhibiting hyperglycemia-induced EMT and fibrosis, thus supporting the therapeutic benefits of this interesting peptide for DR management