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Morshed SA, Davies TF

Adjacent human efficacy in non-growth indications Borasio et al

Subsequently, this impairs IDH1-dependent reductive carboxylation, a tumor cell requirement limited to 3D growth, inducing a decrease in intramitochondrial NADPH, an increase in ROS, and finally mitochondrial supercomplexes assembly disruption, which ultimately impairs cell transformation and tumorigenesis

Only if we show that an improvement in functioning reflects memory as such, and not a change in mood, arousal, attention, executive functioning or anything else, will we have established that an intervention has influenced disease progression