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Although functional loss of SRs can result in hypercholesterolemia, which leads to atherosclerosis and heart disease, it has been shown that when the SR-B2 is exposed to modified LDL, it interacts with TLR4 and TLR6, resulting in NF-B activation and contributing to the inflammatory response associated with atherosclerosis and promoting necrotic lesions of plaques (163, 164)

Notably, more than 99% of the mitochondrial proteome, including all proteins required for mtDNA maintenance, replication and transcription, is encoded by the nuclear DNA (nucDNA) and imported 4 into the organelle

Mao Y, Ma S, Liu C, Liu X, Su M, Li D, et al