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Despite the extensive accumulation of evidence and proposal of multiple pathogenic mechanisms, there is much to learn about how these disorders develop and progress

PRMT5-mediated histone arginine methylation antagonizes transcriptional repression by polycomb complex PRC2

S., Abrao, M

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What is worth to summarize here is the fact that several pro-apoptotic stimuli induce an early extrusion of GSH from cells leading to a widespread mitochondrial damage and to cytochrome c release into the cytosol, the starting condition for apoptotic intrinsic pathway commitment