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We also demonstrate that APR-246 modulates xCT expression in both a p53-dependent and -independent manner

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However, since the interaction between PD-1 and its ligand PD-L1 is the core mechanism of tumor immune escape, Cu can upregulate PD-L1 expression in cancer cells by activating the JAK/STAT signaling pathway and promoting STAT3 and EGFR phosphorylation, thereby promoting tumor growth and immune escape (Qin et al., 2023)

10.1016/j.arr.2022.101790 272 SobralF.SampaioA.FalcoS.QueirozM