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Saturation of the mitochondrial NADH shuttles drives aerobic glycolysis in proliferating cells

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Effect of Ang II on medullary RBF Ang II elicits a paradoxical medullary vasodilatation in normotensive animals, mediated by a secondary activation of vasodilator paracrine agents such as prostaglandins, kinins and NO, rather than to a direct action via AT 2 receptors

It also acts as a potent antioxidant, protecting the brain from oxidative stress and supporting optimal brain function