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While the chemical intermediates of DA oxidation are thoroughly described elsewhere [61], it should be noted that DA quinones have been implicated in mitochondrial dysfunction by forming adducts with proteins such as the subunit of ATP synthase [63] and the E3 ubiquitin ligase Parkin [64], inducing mitochondrial permeability transition [63,65], and inhibiting complex I [66]

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Panda, Berhampur University, India

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