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doi: 10.1016/j.appet.2022.106260 135

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siRNA nanoparticles targeting CaMKII in lesional macrophages improve atherosclerotic plaque stability in mice

Mechanistically, TB-500s parent peptide (T4) drives endothelial cell migration, tubule formation, and angiogenesis, processes essential for granulation tissue and perfusion during healing

p38 mitogen-activated protein (MAP) kinase was also impaired, resulting in the inhibition of CEPB required for adipogenesis and increase in stability of cyclin D1 that induced p21, an inducer of apoptosis and inhibitor of proliferation, and osteocalcin [32]