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Exogenous addition of pyocyanin to the mutant resulted in an increase in biofilm formation and percentage surface coverage (37 7), although not to the level observed in the wild-type biofilm

53 Therapeutic targeting of cardiac proteostasis The recognition that proteostasis failure contributes directly to cardiovascular disease has accelerated the development of therapeutic strategies aimed at restoring protein quality control rather than targeting isolated downstream manifestations

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In contrast, the symbiotic fungus Fusarium foetens ameliorates MASH via its metabolite FF-C1-mediated CerS6 inhibition, reducing ceramides and improving steatosis, inflammation, and fibrosis 55

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