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Taken together, the findings of autoimmunity in families and the plethora of anti-brain antibodies reported for individuals with ASD suggest that in some patients, autoimmune encephalitis and autoantibodies that target the CNS and ENS may be a pathological or exacerbating factor in neuronal and ASD development

Key finding: Reduced oxidative stress markers and inflammatory signaling in neuronal cells neuroprotection beyond skin and wound healing

A critical appraisal of the human data"

Utility of urinary biomarkers to predict delayed graft function after kidney transplantation [abstract]

Interestingly, electroencephalographic recordings often do not fully align with these subjective reports, raising questions about the tools used to measure sedation

In SPMS, characterized by steady deterioration without acute attacks, treatment strategies often involve continuing or altering DMT used during the earlier RRMS phase, such as interferon beta, or considering immune-modulating agents, such as methotrexate, cyclophosphamide, or glucocorticoid pulses [142, 143]