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Anthracyclines Anthracycline drug family discovery is dated in the 1950s with daunorubicin isolation from Streptomyces peucetius ( Anthracycline anti-tumor effect depends on several mechanisms such as apoptosis induction via inhibition of topoisomerase II (TOP2), intercalation into the deoxyribonucleic acid (DNA) leading to an inhibition of macromolecules synthesis, or production of reactive oxygen species (ROS) causing DNA damage or lipid peroxidation ( Anti-tumor effect of anthracyclines is accompanied by dose-dependent cardiac toxicity that has been thoroughly investigated in studies trying to establish a safe dose of anthracyclines ( Cardiotoxicity of DOX clinically presents as both acute and chronic

In dermal wound models, GHK-Cu treatment accelerates wound contraction, stimulates collagen synthesis (types I and III), and promotes the formation of organized granulation tissue

JAMA 218:10151022

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Changes in collagen content of the dermis were assessed by Massons trichrome staining, yielding a blue-violet nucleus

J Aging Phys Act (2019) 27:38491