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In the context of energy perception disorders induced by SUA and mitochondrial dysfunction, when energy perception disorders and mitochondrial damage occur, abnormal energy metabolism and the resultant generation of reactive oxygen species activate a series of stress-sensitive signaling pathways, including the c-Jun N-terminal kinase, protein kinase C, and IKK /NF-B pathways (52, 53)

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Energy metabolism and glutamate-glutamine cycle in the brain: A stoichiometric modeling perspective

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Verapamil block of T-type calcium channels